Böcker i Frontiers in Molecular Biology-serien

Containing a review of current research areas in molecular immunology, this volume includes discussion of immunoglobulin gene rearrangement, the MHC antigens, B-cell activation and the complement system.

This book surveys the latest research into the cytokine network from genomics through signalling pathways and control of the immune response. It does not survey individual cytokines but considers the wider significance of the network and its role in normal and disease states.

Parasitic protozoa cause some of the major infectious diseases of humans and domestic animals. In recent years, new molecular biological techniques have opened up the study of the biology of these parasites. This study covers the recent developments of a rapidly evolving field of research.

The use of developing technologies has revealed the extraordinary degree to which cell cycle control mechanisms have been conserved through eukaryotic evolution. This monograph relates the current scientific understanding of cell cycle control.

Protein-protein recognition is a critical event controlling in a large number of cell processes and therefore is of interest to a large section of the biological community. The purpose of this book is to bring together important concepts and systems in a single volume.

Oncogenes and tumour suppressor genes are two types of genes which are involved in the regulation of the cell cycle and either cause or suppress tumour formation. This volume takes stock of some of the major developments in this critical field. An eminent team of researchers have been commissioned to summarize the current state of their particular areas of expertise.

Plant growth and development is controlled by environmental cues (e.g. light, salinity) that are sensed by the plant via a variety of signal transduction pathways. This book gives an up-to-date summary of the large amount of information that is now available on the processes involved in the communication of plants with their environment.