Summarizes our current understanding of the factors involved in the regulation of transcapillary fluid movement, how fluid movements across the endothelial barrier and through the interstitium and lymphatic vessels influence cell function and behaviour, and the pathophysiology of edema formation.
Reviews the evidence supporting the concept that intrinsic cell survival programmes can be activated by a variety of mildly noxious stimuli or pharmacologic agents to confer protection against the deleterious effects of ischemia/reperfusion.
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